Health and Wellness

How to rid yourself of crusty skin patches triggered by sunlight. It strikes often on the scalp or chest and affects a quarter of those in middle-age. Now doctors reveal the drugs that work – and when you need to worry

It’s been a topic of rabid internet speculation in recent days. The health of Donald Trump was once again called into question on Tuesday, after eagle-eyed viewers caught sight of a vivid red rash on his neck. 

The skin irritation was visible as the US President, 79, gave an update on the war against Iran, prompting debate over its cause, and what treatment he might be undergoing.

A statement issued by Trump’s physician, Dr Sean Barbabella, claimed Mr Trump was using a ‘very common cream on the right side of his neck’ as a ‘preventative treatment’.  The redness, he added, is only expected to last a few weeks. 

Commentators took to social media to float different possibilities for Trump’s diagnosis, which has not been confirmed. 

Some declared the rash to be contact dermatitis caused by dry-cleaning chemicals used on his suits, while others put it down to rosacea – a skin condition the President has been confirmed to suffer, which causes flushing and redness.

Still more suggested the crusty appearance could indicate shingles, a very painful rash of blisters caused by the chickenpox virus.

Experts are speculating that Donald Trump is suffering from actinic keratoses, an unsightly skin condition that increases the risk of skin cancer 

The skin irritation was visible as he delivered an update on the war against Iran , prompting online debate about his health and what treatment he might be undergoing

The skin irritation was visible as he delivered an update on the war against Iran , prompting online debate about his health and what treatment he might be undergoing 

But experts speaking to the Daily Mail confirmed the most likely cause of Mr Trump’s rash is a common skin ailment that will affect roughly a quarter of all adults, particularly in middle age and beyond. 

Actinic keratoses – also known as solar keratoses – are dry, scaly patches of skin that tend to appear on areas frequently exposed to sunlight, such as the face, scalp, ears, neck, backs of the hands and forearms.

Caused by long-term sun exposure, the brown or pink patches often occur on the scalps of bald men or the chests of women.

According to the NHS, the condition is not usually serious. But there is a small risk that, without treatment, the patches can develop into squamous cell carcinoma, a type of skin cancer that begins in the top layer of the skin.

Dr Paul Farrant, consultant dermatologist and director at the Devonshire Clinic, told the Daily Mail: ‘This kind of red very angry rash on the right side of Mr Trump’s neck could definitely be caused by the treatment for actinic keratosis, a precancerous skin condition caused by sun damage.

‘When these creams are applied we would definitely expect long lasting redness and irritation, which looks like what’s going on underneath Trump’s collar.’ 

So, what can you do if you think you’re suffering from the same unsightly skin patches as Donald Trump? 

First, say experts, it’s essential to rule out anything more sinister.

Not all scaly or crusty rashes will be actinic keratosis – and most brown patches on the skin are nothing to worry about, says dermatologist at Dr Ducu Clinics, Dr Anna Andrienko.

Growths that appear waxy and slightly raised, usually ranging in colour from pink to brown to almost black, are likely seborrheic keratoses, or age spots – benign growths affecting half of men and more than a third of women. 

They can grow, but are harmless and don’t need treatment, but can be removed for aesthetic reasons. 

However, if they appear very dark, get them checked to rule out melanoma, the most dangerous type of skin cancer. 

Actinic keratosis, meanwhile, tends to present as rough, gritty patches that feel like sandpaper. 

‘They may be pink, red, skin coloured or slightly brown, and commonly appear on sun exposed areas such as the face, scalp in balding men, ears, forearms and backs of the hands,’ said Dr Andrienko. 

‘It’s crucial to differentiate them from other lesions, such as seborrhoeic keratoses, psoriasis, eczema or early squamous cell carcinoma, which can all look similar. 

‘This can be assessed by a dermatologist who will examine the texture, border, thickness and any signs of ulceration or rapid growth. 

‘In uncertain cases, a biopsy may be required to rule out invasive skin cancer.’ 

Those with fair skin, light eyes and hair and who burn easily rather than tan are all at higher risk for actinic keratosis, says Dr Andrienko. 

The condition also becomes more prevalent with age – those over 40 are more commonly affected, as the condition can be caused by cumulative sun exposure.

Outdoor workers, frequent sunbed users, and people living in sunny climates are at higher risk, as are immunosuppressed patients, such as organ transplant recipients, research shows. 

But whilst it’s common, actinic keratosis isn’t always harmless. 

Skin patches you don't need to worry about are seborrheic keratoses ¿ benign growths affecting half of men and more than a third of women

Skin patches you don’t need to worry about are seborrheic keratoses – benign growths affecting half of men and more than a third of women

More worrisome are actinic keratoses ¿ also called solar keratoses ¿ dry, scaly patches that feel rough, like sandpaper.

More worrisome are actinic keratoses – also called solar keratoses – dry, scaly patches that feel rough, like sandpaper.

In up to five per cent of cases, the condition can develop into squamous cell carcinoma, the most common type of skin cancer – becoming tender, ulcerous or sore.

Luckily, says Dr Andrienko, there are a number of effective therapies that can clear up the condition before it reaches that stage – depending on its severity. 

If you have just one patch of lesions, doctors may recommend a ‘wait and see’ approach, monitoring for any changes in size or quantity. 

But patients with more than one patch, lesions that are painful or itchy, or more than 10 lesions in a patch, are generally recommended to seek treatment.  

If the condition is in its earliest stages, made up of just isolated spots, cryotherapy – where the lesions are frozen off with liquid nitrogen – is often the best option. 

The procedure takes just seconds, and must be done by a dermatologist, who applies liquid nitrogen via a spray gun or cotton-tipped applicator to the skin. 

This is then left to blister, scab and shed within one to three weeks – leaving new, healthy skin. 

Multiple or larger areas of crusty skin spots may require a cream treatment, however. 

One of the most common, says Dr. Conal Perrett, Consultant Dermatologist at The Devonshire Clinic, is a topical chemotherapy treatment commonly prescribed for widespread sun damage called 5-fluorouracil cream.

The cream works by causing inflammation that, in turn, destroys the pre-cancerous cells that form in actinic keratosis, but leaves the underlying healthy tissue untouched. 

It is usually applied daily for up to four weeks, although some patients need a second round to completely eradicate the lesion, otherwise it grows back. 

It can also cause uncomfortable itching as well as weeping and red skin rashes, similar to that on President Trump’s neck.

‘During treatment, it is entirely expected for the skin to become red, inflamed, sore and sometimes crusted,’ added Dr Perrett. 

‘In fact, visible redness and irritation are generally signs that the medication is working.’

A bright red, inflamed patch confined to one area, such as one side of the neck like that seen on President Trump, he went on, would be consistent with topical treatments for actinic keratosis. 

Said Dr Perrett: ‘Without direct clinical assessment, it would not be possible to confirm the cause of any individual’s skin changes.

‘However, the description of preventative treatment with a commonly used topical cream, associated with short-term use and several weeks of visible redness, would be broadly consistent with standard management of actinic keratoses.’

Tirbanibulin works by stopping pre-cancerous cells from dividing and spreading on sun-damaged skin, and is applied once daily for five days

Tirbanibulin works by stopping pre-cancerous cells from dividing and spreading on sun-damaged skin, and is applied once daily for five days

A newer cream for actinic keratosis, rolled out for use on large patches of the head and neck by the NHS in 2024, is tirbanibulin. 

Like 5-fluorouracil, the cream works by stopping pre-cancerous cells from dividing and spreading on sun-damaged skin. 

While just as effective as 5-fluorouracil, tirbanibulin works far more quickly – applied once daily for just five days.

Photodynamic therapy is another option for larger areas on the face or scalp, says Dr Andrienko.

The treatment involves applying a photosensitising cream to the affected area, which is then activated by either a red light lamp, or natural daylight to destroy abnormal cells. 

After about two weeks, scabs form and fall off, leaving healthy skin behind. 

But for everyone, says Dr Andrienko, prevention is key. 

‘Daily broad spectrum SPF, protective clothing, and regular skin checks are essential to prevent actinic keratosis from developing, particularly for high risk individuals,’ she said. 

Does actinic keratosis always need treatment?  

Doctors in the UK may be overtreating actinic keratoses that would likely never have caused problems, claims a new study. 

Patients with no history of skin cancer, have fewer than 10 lesions, and are not immunocompromised should not be immediately offered treatment, concluded a paper published this week, from researchers at the University of Michigan. 

Despite actinic keratoses being amongst the most common form of skin lesion, very few actually progress into cancer, they wrote.

Treatment, meanwhile, can cause debilitating side effects – including pain, blistering, pigment changes and scarring. 

Research shows that while common treatments such as cryotherapy and topical creams often result in the temporary clearance of actinic keratosis lesions, the recurrence rate is the same, one year later, as in those who received no treatment at all. 

Instead, the researchers noted, doctors may feel pressure to treat lesions due to fears that it will progress to cancer, as well as pressure from patients who find them unsightly.

In the US, where physicians may also have a financial incentive to treat the condition, treatment is even more likely to be offered, they explained.

 ‘These structural forces make treatment the path of least resistance, even when monitoring may offer equal or greater value,’ explained lead researcher Dr Bingjie Pecha. 

Instead, they concluded, active surveillance, rather than immediate treatment, should be the first port of call for patients with low-risk disease. 

‘This approach may reduce unnecessary intervention, preserve quality of life, and allow timely intervention if disease progresses,’ said Dr Pecha. 

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